Contents

Disclaimer

Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct. Readers must therefore always check the product information and clinical procedures with the most up to date published product information and data sheets provided by the manufacturers and the most recent codes of conduct and safety regulations. The authors and the publishers do not accept responsibility or legal liability for any errors in the text or for the misuse or misapplication of material in this work. Except where otherwise stated, drug dosages and recommendations are for the non-pregnant adult who is not breastfeeding.

Show Summary Details
Page of

Tendinopathies and enthesopathies 

Tendinopathies and enthesopathies
Chapter:
Tendinopathies and enthesopathies
Author(s):

Thomas Crisp

DOI:
10.1093/med/9780199674107.003.0045
Page of

PRINTED FROM OXFORD MEDICINE ONLINE (www.oxfordmedicine.com). © Oxford University Press, 2021. All Rights Reserved. Under the terms of the licence agreement, an individual user may print out a PDF of a single chapter of a title in Oxford Medicine Online for personal use (for details see Privacy Policy and Legal Notice).

date: 02 March 2021

Adult ligaments and tendons are very similar collagenized structures, composed of largely type 1 collagen in tight bands, with very little type III collagen. The fluid between the fibrils contains proteoglycans and a small number of elastic fibres. The number and type of collagen fibrils correlates closely with the tensile strength. The tendon is able to stretch up to about 4% without any loss of structure and tearing occurs after this point up to complete rupture at about 10% stretch. Constant turnover of collagen is carried out by tenocytes in the tendon and changes occur to this process in tendinopathy. Such changes may cause more water to be held in the tendon, thereby contributing to swelling of the tendon. Metalloproteinases, which show increased levels in painful tendinopathy, play an undetermined role in this degradation process. The increase in neuropeptides in degenerate tendons is also thought to play a part.

Access to the complete content on Oxford Medicine Online requires a subscription or purchase. Public users are able to search the site and view the abstracts for each book and chapter without a subscription.

Please subscribe or login to access full text content.

If you have purchased a print title that contains an access token, please see the token for information about how to register your code.

For questions on access or troubleshooting, please check our FAQs, and if you can't find the answer there, please contact us.