- Preface
- Contributors
- Symbols and Abbreviations
- Classes of recommendations and levels of evidence
- Amendments and Updates
- Chapter 1.1 Hypertension
- Chapter 1.2 Dyslipidaemia
- Chapter 1.3 Metabolic syndrome and diabetes
- Chapter 1.4 Thrombosis
- Chapter 2.1 Acute coronary syndrome: STEMI and NSTEMI
- Chapter 2.2 Chronic stable angina
- Chapter 2.3 Coronary artery spasm and microvascular angina
- Chapter 2.4 Takotsubo syndrome
- Chapter 3.1 Heart failure
- Chapter 3.2 Heart transplantation
- Chapter 4.1 Atrial fibrillation
- Chapter 4.2 Supraventricular (narrow complex) tachycardias
- Chapter 4.3 Ventricular arrhythmias
- Chapter 4.4 Bradyarrhythmias
- Chapter 4.5 Syncope
- Chapter 5.1 Valvular heart disease
- Chapter 5.2 Myocarditis and pericardial syndromes
- Chapter 5.3 Cardiomyopathy
- Chapter 5.4 Pulmonary hypertension
- Chapter 6.1 Kidney disease
- Chapter 6.2 Pregnancy and lactation
- Chapter 6.3 Liver disease
- Chapter 7.1 Major drug interactions
- Chapter 8.1 Cardiovascular drugs—from A to Z
- Chapter 9.1 Non-cardiac drugs affecting the heart—from A to Z
- Index
(p. 209) Supraventricular (narrow complex) tachycardias
- Chapter:
- (p. 209) Supraventricular (narrow complex) tachycardias
- Author(s):
Julio Martí-Almor
- DOI:
- 10.1093/med/9780198759935.003.0012_update_001
Update:
Tables 4.2.2, 4.2.3, and 4.2.4 extensively updated
Further reading added
Supraventricular tachycardia (SVT) includes all tachycardia rhythms (more than 100 bpm), of which the mechanism involves the His Bundle or tissues above it. Usually, these tachyarrhythmias present a narrow QRS complex (except in the presence of a bundle branch block or a manifest accessory pathway which conducts anterogradely to cause a ventricular pre-excitation pattern on the electrocardiogram). This chapter only focuses on narrow QRS complex SVT involving the atrial tissue, the atrioventricular node, and accessory pathways. Atrial fibrillation and atrial flutter are excluded because they are specifically covered in other chapters. The role of antiarrhythmic drugs in SVTs is mainly restricted to acute situations because ablative therapy has surrogate pharmacotherapy and ablation is considered the best long-term treatment for most cases. Nevertheless, it is very important to know how to deal with antiarrhythmic drugs in emergency situations.
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- Preface
- Contributors
- Symbols and Abbreviations
- Classes of recommendations and levels of evidence
- Amendments and Updates
- Chapter 1.1 Hypertension
- Chapter 1.2 Dyslipidaemia
- Chapter 1.3 Metabolic syndrome and diabetes
- Chapter 1.4 Thrombosis
- Chapter 2.1 Acute coronary syndrome: STEMI and NSTEMI
- Chapter 2.2 Chronic stable angina
- Chapter 2.3 Coronary artery spasm and microvascular angina
- Chapter 2.4 Takotsubo syndrome
- Chapter 3.1 Heart failure
- Chapter 3.2 Heart transplantation
- Chapter 4.1 Atrial fibrillation
- Chapter 4.2 Supraventricular (narrow complex) tachycardias
- Chapter 4.3 Ventricular arrhythmias
- Chapter 4.4 Bradyarrhythmias
- Chapter 4.5 Syncope
- Chapter 5.1 Valvular heart disease
- Chapter 5.2 Myocarditis and pericardial syndromes
- Chapter 5.3 Cardiomyopathy
- Chapter 5.4 Pulmonary hypertension
- Chapter 6.1 Kidney disease
- Chapter 6.2 Pregnancy and lactation
- Chapter 6.3 Liver disease
- Chapter 7.1 Major drug interactions
- Chapter 8.1 Cardiovascular drugs—from A to Z
- Chapter 9.1 Non-cardiac drugs affecting the heart—from A to Z
- Index