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OPML Neuropathic Pain$
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OPML Neuropathic Pain (2 ed.)

Edited by Michael Bennett

Publisher:
Oxford University Press
Print Publication Date:
Dec 2010
Print ISBN-13:
9780199563678
Published to Oxford Medicine:
May 2011
DOI:
10.1093/med/9780199563678.001.0001

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Oxford University Press makes no representation, express or implied, that the drug dosages in this book are correct. Readers must therefore always check the product information and clinical procedures with the most up to date published product information and data sheets provided by the manufacturers and the most recent codes of conduct and safety regulations. The authors and the publishers do not accept responsibility or legal liability for any errors in the text or for the misuse or misapplication of material in this work. Except where otherwise stated, drug dosages and recommendations are for the non-pregnant adult who is not breastfeeding.

Theories, history, and current taxonomy

Chapter:
Theories, history, and current taxonomy
DOI:
10.1093/med/9780199563678.003.0001

• Pain is a complex experience of somatic mechanisms and psychological influences (affective and cognitive) and, as such, is always subjective in nature

• The nervous system is capable of substantial plasticity; it can change structure and function in response to development, injury, or experience

• Nociceptive or inflammatory pain arises from normal activation of pain pathways

• Neuropathic pain is pain arising as a direct consequence of a lesion or disease affecting the somatosensory system (abnormal activation of pain pathways)

• Pain is usually classified by mechanism (nociceptive or neuropathic), context (physiological or pathological), and location (somatic, visceral, etc.).

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