- Understanding Depression
- Chapter 1 Experiencing depression
- Chapter 2 Depression in the medically ill
- Chapter 3 Explaining depression: neuroscience is not enough, evolution is essential
- Chapter 4 Evolutionary genetics of affective disorders
- Chapter 5 Genetic factors in stress and major depression
- Chapter 6 Bipolar disorder and unipolar depression: what is the genetic relationship?
- Chapter 7 The significance of dysregulated basal glucocorticoid pulsatility in affective disorders
- Chapter 8 Early life programming of affective function
- Chapter 9 Modelling depression by GR mutant animals?
- Chapter 10 Cytokines and depression: experimental evidence and intermediate mechanisms
- Chapter 11 Do depression, stress, sleep disruption, and inflammation alter hippocampal apoptosis and neurogenesis?
- Chapter 12 The role of the medial prefrontal cortex in mediating resistance and vulnerability to the impact of adverse events
- Chapter 13 Is it all monoamines?
- Chapter 14 The role of affective processing in vulnerability to and resilience against depression
- Chapter 15 PTSD and HPA axis: same hormones, different disorders
- Chapter 16 Neuroendocrine and immune crosstalk in major depression
- Chapter 17 Neuropsychiatric effects of IFN-alpha: relevance to depression
- Chapter 18 Depression and coronary heart disease
- Chapter 19 Does depression induce pain and fatigue?
- Chapter 20 Explaining the association between depression and mortality: a life course epidemiology approach
- Chapter 21 Depression: unipolar or bipolar, what’s the difference (and what does it matter anyway)?
- Chapter 22 Can neuroimaging help distinguish bipolar depression from major depressive disorder?
- Chapter 23 Pharmacogenetics of antidepressant response
- Chapter 24 Can the EEG be used to predict antidepressant response?
- Chapter 25 Can we use magnetic/electric fields to help in treatment-resistant depression?
- Chapter 26 Antiglucocorticoids in the treatment of affective disorders: from preclinical to clinical studies
- Chapter 27 How can we use current knowledge to improve antidepressant treatments?
(p. 17) Explaining depression: neuroscience is not enough, evolution is essential
- (p. 17) Explaining depression: neuroscience is not enough, evolution is essential
Randolph M. Nesse
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